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April 2014- AXA professor of Risk prediction in age-related diseases
September 2009- ICREA Research Professor
‘DNA spellchecker’ means that our genes aren’t all equally likely to mutate and cause disease (23/02/2015)
A study that examined 17 million mutations in the genomes of 650 cancer patients concludes that large differences in mutation rates across the human genome are caused by the DNA repair machinery.
Age-related diseases and adversary risk analysis are the two new AXA Research Fund in Spain research chairs (04/04/2014)
The AXA Research Fund has created the permanent AXA Chair in Risk prediction in age-related diseases, given to Ben Lehner, from the Centre for Genomic Regulation (CRG); and...
Ben Lehner awarded a Consolidator Grant of the European Research Council (15/01/2014)
Ben Lehner, head of the Genetic Systems research group and ICREA research professor at the Centre for Genomic Regulation (CRG), in Barcelona, has just been awarded a Consolidator Grant from the European Research Council (ERC), for his project "Individual robustness in development and c
The effects of genetic variation in space and time (09/01/2014)
CRG researchers describe in a paper published in Nature that many genetic differences between individuals influence the dynamic patterns of how genes are switched on or off during development. The research will contribute to the understanding of how the differences in our genomes affect dynamic proc
Ben Lehner, receives the Eppendorf Award for Young Investigators (11/06/2013)
Ben Lehner has won the 2013 Eppendorf Young Investigator Award. The official Award ceremony took place at the EMBL Advanced Training Centre in Heidelberg, Germany, on June 6, 2013.
To what extent is it possible to predict the phenotypic differences amongst individuals from their completely sequenced genomes? We use model organisms (yeast, worms) and computational analyses to understand when you can, and why you cannot, predict the biology of an individual from their genome sequence. Individuals are thankfully all different, and our aim is to understand how these differences originate in the interactions between genetic, environmental, life history, parental and stochastic sources of variation.
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Puromycin vector sequences